最新外科急诊创伤(英文)-烧伤精品课件.ppt
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1、ScenarioParamedic is called to the scene of a structure fire. FD has removed a victim from the house. BSIScene safe1 patientA/C standbyFD/ PD on sceneNow what?LayerslEpidermislDermislSubcutaneoslUnderlying StructureslFascialNerveslTendonslLigamentslMuscleslOrgansAnatomy & Physiology of the SkinFunct
2、ionProtection Regulation Prevention SensoryEpidermisOuter, thinner layerConsists of dead keratinized cellsProtects dehydration trauma light infectionDermisGel like matrixConsists of collagen and elastinContains blood vessels, lymphatics, sweat glands, hair follicles, sensory fibersSubcutaneousConnec
3、tive tissueAdipose tissue cushioning insulationCausesThermalElectricalChemicalRadiation ThermalMajority flame scald contact with hot objectsChild with burns from a scaldDetermining Severity1st degree2nd degree3rd degree(4th degree)Depth of BurnSuperficial BurnPartial Thickness BurnFull Thickness Bur
4、nFirst DegreeSuperficial involve only epidermisLocal pain and rednessNo blistering presentHeal spontaneously 2-5 days without scarringNot included when calculating % TBSABurn DepthSuperficial Burn:1st Degree BurnlSigns & SymptomslReddened skinlPain at burn sitelInvolves only epidermisSecond DegreeIn
5、volve epidermis and dermisPartial thickness superficial partial thickness red, painful, blistered deep partial thickness pale, mottledVery painfulInfection may evolve into 3rd degreeBurn DepthPartial-Thickness Burn: 2nd Degree BurnlSigns & SymptomslIntense painlWhite to red skinlBlisterslInvolves ep
6、idermis & dermisThird DegreeInvolve epidermis, dermis, subcutaneous tissueWhite, waxy, red, brown, leatheryDry and painless(muscle and bone)Burn DepthFull-Thickness Burn: 3rd Degree BurnlSigns & SymptomslDry, leathery skin (white, dark brown, or charred)lLoss of sensation (little pain)lAll dermal la
7、yers/tissue may be involvedFourth DegreeInclude involvement of muscle and boneCharred in appearancePainlessPathophysiologyLocal changes- 111F produce injuryArea of DamageZone of coagulationZone of stasisZone of hyperemiaJacksons Theory of Thermal WoundslZone of CoagulationlArea in a burn nearest the
8、 heat source that suffers the most damage as evidenced by clotted blood and thrombosed blood vesselslZone of StasislArea surrounding zone of coagulation characterized by decreased blood flow.lZone of HyperemialPeripheral area around burn that has an increased blood flow.Jacksons Theory of Thermal Wo
9、undsZone of HyperemiaZone of StasisZone of CoagulationZone of CoagulationCentral area of burnNecrotic from time of exposureZone of StasisModerate degree of insultDecreased tissue perfusionVascular damage/ leakageMay progress to necrosis 24-48 hoursZone of HyperemiaVasodilationInflammationViable tiss
10、ueBodys Response to BurnsEmergent Phase (Stage 1)lPain responselCatecholamine releaselTachycardia, Tachypnea, Mild Hypertension, Mild AnxietyFluid Shift Phase (Stage 2)lLength 18-24 hourslBegins after Emergent PhaselReaches peak in 6-8 hourslDamaged cells initiate inflammatory responselIncreased blo
11、od flow to cellslShift of fluid from intravascular to extravascular spacelMASSIVE EDEMAl“Leaky CapillariesSystemic ChangesMassive release of inflammatory mediatorsProduce vasoconstriction/ dilationIncreased capillary permeabilityEdemaFluid ShiftsInitial decrease blood flow to burned areaFollowed by
12、increased arterial vasodilationRelease of vasoactive substance resulting in increased capillary permeability and edemaCardiovascularLoss of plasma volumeIncreased peripheral vascular resistanceDecreased cardiac output decreased blood volume decreased venous return increased blood viscosity decreased
13、 contractilityRenalDecrease circulating plasmaIncrease hematocritDecreased CO decreased renal blood flow oliguria acute renal failureGastrointestinalDecreased gastrointestinal blood flowIncreased mucosal hemorrhage 20% ileusImmune SystemDepressed immune function 20% directly proportional to burn siz
14、esepsisBodys Response to BurnsHypermetabolic Phase (Stage 3)lLast for days to weekslLarge increase in the bodys need for nutrients as it repairs itselfResolution Phase (Stage 4)lScar formationlGeneral rehabilitation and progression to normal functionHypermetabolismFollowing severe burn and resuscita
15、tion tachycardia increased CO increased O2 demand massive proteolysis & lipolysis severe nitrogen lossSystemic ComplicationsHypothermialDisruption of skin and its ability to thermoregulateHypovolemialShift in proteins, fluids, and electrolytes to the burned tissuelGeneral electrolyte imbalanceEschar
16、lHard, leathery product of a deep full thickness burnlDead and denatured skinSystemic ComplicationsInfectionlGreatest risk of burn is infectionOrgan FailurelRelease of myoglobinSpecial FactorslAge & HealthPhysical AbuselElderly, Infirm or YoungCritical Burn AreasFace HandsFeetGroinJointsCircumfrenti
17、alInhalation InjuriesLeading cause of death Closed space incident Presence of heavy smoke History of unconsciousnessBurns, thermal. Partial- and full-thickness burns from structure fire. Note facial involvement.Inhalation InjuryToxic InhalationlSynthetic resin combustionlCyanide & Hydrogen SulfidelS
18、ystemic poisoninglMore frequent than thermal inhalation burnCarbon Monoxide PoisoninglColorless, odorless, tasteless gaslByproduct of incomplete combustion of carbon productslSuspect with faulty heating unitl200 x greater affinity for hemoglobin than oxygenlHypoxemia & HypercarbiaOther EvidenceFacia
19、l burnsProfuse secretionsCarbonaceous sputumLacrimationSinged nasal hairHoarsenessWheezingStridorEdemaHypoxemiaTachycardiaInhalation InjuryAirway Thermal BurnlSupraglottic structures absorb heat and prevent lower airway burnslMoist mucosa lining the upper airwaylInjury is common from superheated ste
20、amlRisk FactorslStanding in the burn environmentlScreaming or yelling in the burn environmentlTrapped in a closed burn environmentlSymptomslStridor or “Crowing” inspiratory soundslSinged facial and nasal hairlBlack sputum or facial burnslProgressive respiratory obstruction and arrest due to swelling
21、Types of InjuriesCarbon monoxide poisoningInjury above glottisInjury below glottisCO PoisoningAffinity for Hgb 200-250X than O2Cherry red only present at levels 40%+N,+V, HA, decreased LOC, weakness, tachypnea, tachycardia False pulse oximetry reading 100% O2 time for elimination 40 min21% O2 time e
22、limination 250 minutesCarboxyhemoglobinNormal- 0Smokers, truck drivers in heavy traffic- 1515-40%- neurological dysfunction weakness, dizziness, +N, +V, HA40-60%- obtunded severe decreased LOCConsider hyperbaric therapy- 25-40%Injury Above GlottisThermal, chemicalRequire early intubationSeverely hyp
23、ovolemicInjury Below GlottisUsually chemicalRepiratory distressRequire early intubationARDSMSOFEstimating % BSA BurnedRule of palmsRule of ninesBody Surface AreaRule of NineslBest used for large surface areaslExpedient tool to measure extent of burnRule of PalmslBest used for burns 10% BSARules of N
24、ines184.59194.5184.54.5994.5994.574.574.5718181Rule of PalmsA burn equivalent to the size of the patients hand is equal to 1% body surface area (BSA)TreatmentStop the burnABCsEstimate % BSA burnedCool burnPrevent hypothermia & infectionPain controlAirwayO2 on ALL patients Acute pulmonary insufficien
25、cy Pulmonary edema 2-3 days Bronchopneumonia 5-7 daysConsider intubation Sx/ liklihood of impending airway obstructionCirculationFluid replacement critical to survivalTissue destruction results in increased capillary permeabilityProfound fluid loss from the intravascular spaceLarge amounts fluid los
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